polyglutamine

TATA-box binding protein ; Homo sapiens







28 Article(s)
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1 34906452 Digenic inheritance of STUB1 variants and TBP polyglutamine expansions explains the incomplete penetrance of SCA17 and SCA48. 2022 Jan 2
2 30707359 EvoPPI 1.0: a Web Platform for Within- and Between-Species Multiple Interactome Comparisons and Application to Nine PolyQ Proteins Determining Neurodegenerative Diseases. 2019 Mar 2
3 30760647 Shaoyao Gancao Tang (SG-Tang), a formulated Chinese medicine, reduces aggregation and exerts neuroprotection in spinocerebellar ataxia type 17 (SCA17) cell and mouse models. 2019 Feb 13 2
4 31317427 Molecular Mechanisms and Therapeutics for SCA17. 2019 Oct 5
5 29427105 Spinocerebellar Ataxia Type 17 (SCA17). 2018 2
6 29936316 The indole compound NC009-1 inhibits aggregation and promotes neurite outgrowth through enhancement of HSPB1 in SCA17 cells and ameliorates the behavioral deficits in SCA17 mice. 2018 Jul 1
7 30473770 Recent advances in understanding dominant spinocerebellar ataxias from clinical and genetic points of view. 2018 1
8 28821675 Synergistic Toxicity of Polyglutamine-Expanded TATA-Binding Protein in Glia and Neuronal Cells: Therapeutic Implications for Spinocerebellar Ataxia 17. 2017 Sep 20 8
9 27520369 Conformational modulation mediated by polyglutamine expansion in CAG repeat expansion disease-associated proteins. 2016 Sep 16 1
10 24510561 [Advance in research on spinocerebellar ataxia 17]. 2014 Feb 1
11 25342886 Indole and synthetic derivative activate chaperone expression to reduce polyQ aggregation in SCA17 neuronal cell and slice culture models. 2014 9
12 22664922 Regulation of BACE1 by miR-29a/b in a cellular model of Spinocerebellar Ataxia 17. 2012 Jun 3
13 21653638 A Drosophila model of the neurodegenerative disease SCA17 reveals a role of RBP-J/Su(H) in modulating the pathological outcome. 2011 Sep 1 4
14 20226542 Human ataxias: a genetic dissection of inositol triphosphate receptor (ITPR1)-dependent signaling. 2010 May 1
15 19285969 A splice variant of the TATA-box binding protein encoding the polyglutamine-containing N-terminal domain that accumulates in Alzheimer's disease. 2009 May 1 1
16 18218637 Polyglutamine expansion reduces the association of TATA-binding protein with DNA and induces DNA binding-independent neurotoxicity. 2008 Mar 28 1
17 18418687 Spinocerebellar ataxia 17 (SCA17) and Huntington's disease-like 4 (HDL4). 2008 2
18 17868456 HAP1 can sequester a subset of TBP in cytoplasmic inclusions via specific interaction with the conserved TBP(CORE). 2007 Sep 14 6
19 17994014 Polyglutamine domain modulates the TBP-TFIIB interaction: implications for its normal function and neurodegeneration. 2007 Dec 4
20 15916858 TATA-binding protein in neurodegenerative disease. 2005 2
21 15989694 Spinocerebellar ataxia type 17: report of a family with reduced penetrance of an unstable Gln49 TBP allele, haplotype analysis supporting a founder effect for unstable alleles and comparative analysis of SCA17 genotypes. 2005 Jul 1 2
22 15225551 Cellular toxicity of polyglutamine expansion proteins: mechanism of transcription factor deactivation. 2004 Jul 2 2
23 12758065 Molecular investigation of TBP allele length: a SCA17 cellular model and population study. 2003 Jun 3
24 12891385 SCA17 caused by homozygous repeat expansion in TBP due to partial isodisomy 6. 2003 Aug 1
25 11448935 SCA17, a novel autosomal dominant cerebellar ataxia caused by an expanded polyglutamine in TATA-binding protein. 2001 Jul 1 1
26 12235815 [SCA17, a novel polyglutamine disease caused by the expansion of polyglutamine tracts in TATA-binding protein]. 2001 Dec 1
27 10484774 A neurological disease caused by an expanded CAG trinucleotide repeat in the TATA-binding protein gene: a new polyglutamine disease? 1999 Oct 2
28 8886170 Analysis of polyglutamine-coding repeats in the TATA-binding protein in different human populations and in patients with schizophrenia and bipolar affective disorder. 1996 Sep 20 3