N-Methylaspartate

tumor necrosis factor ; Mus musculus







15 Article(s)
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1 31260687 Endoplasmic reticulum stress contributes to NMDA-induced pancreatic β-cell dysfunction in a CHOP-dependent manner. 2019 Sep 1 1
2 28087150 Apelin protects against NMDA-induced retinal neuronal death via an APJ receptor by activating Akt and ERK1/2, and suppressing TNF-α expression in mice. 2017 Jan 3
3 28356707 Epigenetic intervention with a BET inhibitor ameliorates acute retinal ganglion cell death in mice. 2017 1
4 27371735 Tumor necrosis factor regulates NMDA receptor-mediated airway smooth muscle contractile function and airway responsiveness. 2016 Aug 1 3
5 27791020 Essential protective role of tumor necrosis factor receptor 2 in neurodegeneration. 2016 Oct 25 1
6 25613943 Activation of liver X receptor protects inner retinal damage induced by N-methyl-D-aspartate. 2015 Jan 22 1
7 24283717 Deletion of thioredoxin-interacting protein preserves retinal neuronal function by preventing inflammation and vascular injury. 2014 Mar 2
8 23094196 Rapamycin Augments the NMDA-Mediated TNF Suppression of MRSA-Stimulated RAW264.7 Murine Macrophages. 2012 5
9 21159431 TNF-α contributes to spinal cord synaptic plasticity and inflammatory pain: distinct role of TNF receptor subtypes 1 and 2. 2011 Feb 2
10 21266054 Ketamine inhibits tumor necrosis factor secretion by RAW264.7 murine macrophages stimulated with antibiotic-exposed strains of community-associated, methicillin-resistant Staphylococcus aureus. 2011 Jan 25 2
11 20383154 Resolvins RvE1 and RvD1 attenuate inflammatory pain via central and peripheral actions. 2010 May 1
12 19994584 [Mechanism of N-methyl-D-aspartate-induced retinal ganglion cell death]. 2009 Nov 1
13 17241124 Tumor necrosis factor alpha stimulates NMDA receptor activity in mouse cortical neurons resulting in ERK-dependent death. 2007 Mar 5
14 17266736 Pitavastatin prevents NMDA-induced retinal ganglion cell death by suppressing leukocyte recruitment. 2007 Feb 1
15 14622092 Nicotine-induced neuroprotection against N-methyl-D-aspartic acid or beta-amyloid peptide occur through independent mechanisms distinguished by pro-inflammatory cytokines. 2003 Dec 3