Title : Mutation of a major keratin phosphorylation site predisposes to hepatotoxic injury in transgenic mice.

Pub. Date : 1998 Dec 28

PMID : 9864372






2 Functional Relationships(s)
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1 However, exposure of S52A-expressing mice to the hepatotoxins, griseofulvin or microcystin, which are associated with K18 ser52 and other keratin phosphorylation changes, resulted in more dramatic hepatotoxicity as compared with WT K18-expressing mice. microcystin keratin 18 Mus musculus
2 However, exposure of S52A-expressing mice to the hepatotoxins, griseofulvin or microcystin, which are associated with K18 ser52 and other keratin phosphorylation changes, resulted in more dramatic hepatotoxicity as compared with WT K18-expressing mice. microcystin keratin 18 Mus musculus