Title : Metabolomics analysis of gut barrier dysfunction in a trauma-hemorrhagic shock rat model.

Pub. Date : 2019 Jan 31

PMID : 30393232






4 Functional Relationships(s)
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1 Under the induction of THS, gut barrier failure was characterized by injury of permeability and mucus layer, which were companied by the decreased expression of zonula occludens-1 in the colon and increased levels of inflammatory factors including tumor necrosis factor-alpha, interferon-gamma, interleukin (IL)-6, and IL-1beta in the serum. THYMIDINE-5'-(DITHIO)PHOSPHATE tumor necrosis factor Rattus norvegicus
2 Under the induction of THS, gut barrier failure was characterized by injury of permeability and mucus layer, which were companied by the decreased expression of zonula occludens-1 in the colon and increased levels of inflammatory factors including tumor necrosis factor-alpha, interferon-gamma, interleukin (IL)-6, and IL-1beta in the serum. THYMIDINE-5'-(DITHIO)PHOSPHATE interferon gamma Rattus norvegicus
3 Under the induction of THS, gut barrier failure was characterized by injury of permeability and mucus layer, which were companied by the decreased expression of zonula occludens-1 in the colon and increased levels of inflammatory factors including tumor necrosis factor-alpha, interferon-gamma, interleukin (IL)-6, and IL-1beta in the serum. THYMIDINE-5'-(DITHIO)PHOSPHATE interleukin 6 Rattus norvegicus
4 Under the induction of THS, gut barrier failure was characterized by injury of permeability and mucus layer, which were companied by the decreased expression of zonula occludens-1 in the colon and increased levels of inflammatory factors including tumor necrosis factor-alpha, interferon-gamma, interleukin (IL)-6, and IL-1beta in the serum. THYMIDINE-5'-(DITHIO)PHOSPHATE interleukin 1 beta Rattus norvegicus