Title : CD38-mediated Inhibition of Bruton's Tyrosine Kinase in Macrophages Prevents Endotoxemic Lung Injury.

Pub. Date : 2022 Feb

PMID : 34706199






2 Functional Relationships(s)
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1 Cd38 / macrophages displayed markedly increased activation of Btk, NF-KappaB, and NLRP3 whereas in vivo administration of the Btk inhibitor ibrutinib (a FDA approved drug) prevented augmented TLR4-induced inflammatory lung injury seen in Cd38 / mice. ibrutinib Bruton agammaglobulinemia tyrosine kinase Mus musculus
2 Cd38 / macrophages displayed markedly increased activation of Btk, NF-KappaB, and NLRP3 whereas in vivo administration of the Btk inhibitor ibrutinib (a FDA approved drug) prevented augmented TLR4-induced inflammatory lung injury seen in Cd38 / mice. ibrutinib Bruton agammaglobulinemia tyrosine kinase Mus musculus