Title : Increased oral sodium chloride intake in humans amplifies selectively postprandial GLP-1 but not GIP, CCK, and gastrin in plasma.

Pub. Date : 2020 Aug

PMID : 32770661






4 Functional Relationships(s)
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1 Human studies have demonstrated that physiologically relevant changes in circulating glucagon-like peptide-1 (GLP-1) elicit a rapid increase in renal sodium excretion when combined with expansion of the extracellular fluid volume. Sodium glucagon Homo sapiens
2 Human studies have demonstrated that physiologically relevant changes in circulating glucagon-like peptide-1 (GLP-1) elicit a rapid increase in renal sodium excretion when combined with expansion of the extracellular fluid volume. Sodium glucagon Homo sapiens
3 This study was designed to investigate the hypothesis that the postprandial GLP-1 plasma concentration is sensitive to the sodium content in the meal. Sodium glucagon Homo sapiens
4 Sodium intake in addition to a glucose load selectively amplifies the postprandial GLP-1 plasma concentration. Sodium glucagon Homo sapiens