Title : Dietary therapy restores glutamatergic input to orexin/hypocretin neurons after traumatic brain injury in mice.

Pub. Date : 2018 Mar 1

PMID : 29315422






2 Functional Relationships(s)
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1 TBI was speculated to reduce orexin neuronal activity through decreased regional excitatory (glutamate) and/or increased inhibitory (GABA) input. Glutamic Acid hypocretin Mus musculus
2 Conclusions: These results suggest TBI compromises orexin neuron function via decreased glutamate density and highlight BCAA supplementation as a potential therapy to restore glutamate density to orexin neurons. Glutamic Acid hypocretin Mus musculus