Title : Benzyl isothiocyanate promotes apoptosis of oral cancer cells via an acute redox stress-mediated DNA damage response.

Pub. Date : 2016 Nov

PMID : 27693243






1 Functional Relationships(s)
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1 Notably, BITC-induced apoptosis was associated with reduced Mcl-1 and Bcl-2 expression, diminished mitochondrial membrane potential (DeltaPsim), and increased PARP cleavage. benzyl isothiocyanate BCL2 apoptosis regulator Homo sapiens