Title : Endothelial acyl-CoA synthetase 1 is not required for inflammatory and apoptotic effects of a saturated fatty acid-rich environment.

Pub. Date : 2013 Feb

PMID : 23241406






2 Functional Relationships(s)
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1 The objective of this study was to investigate the role of ACSL1, an ACSL isoform previously shown to mediate inflammatory effects in myeloid cells, in regulating endothelial cell responses to a saturated fatty acid-rich environment in vitro and in vivo. Fatty Acids acyl-CoA synthetase long-chain family member 1 Mus musculus
2 Forced ACSL1 overexpression exacerbated the effects of saturated fatty acids on apoptosis and endoplasmic reticulum stress. Fatty Acids acyl-CoA synthetase long-chain family member 1 Mus musculus