Title : eNOS protects from atherosclerosis despite relevant superoxide production by the enzyme in apoE mice.

Pub. Date : 2012

PMID : 22291917






5 Functional Relationships(s)
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1 eNOS protects from atherosclerosis despite relevant superoxide production by the enzyme in apoE mice. Superoxides nitric oxide synthase 3, endothelial cell Mus musculus
2 Here, we evaluate how endothelial nitric oxide synthase (eNOS) modulates leukocyte/endothelial- (L/E) and platelet/endothelial- (P/E) interactions in atherosclerosis and the production of nitric oxide (NO) and superoxide by the enzyme. Superoxides nitric oxide synthase 3, endothelial cell Mus musculus
3 Here, we evaluate how endothelial nitric oxide synthase (eNOS) modulates leukocyte/endothelial- (L/E) and platelet/endothelial- (P/E) interactions in atherosclerosis and the production of nitric oxide (NO) and superoxide by the enzyme. Superoxides nitric oxide synthase 3, endothelial cell Mus musculus
4 Pharmacological inhibition and genetic deletion of eNOS reduced vascular superoxide production, indicating uncoupling of the enzyme in apoE(-/-) vessels. Superoxides nitric oxide synthase 3, endothelial cell Mus musculus
5 CONCLUSION: Overt plaque formation, increased vascular inflammation and L/E- interactions are associated with significant reduction of superoxide production in apoE(-/-)/eNOS(-/-) vessels. Superoxides nitric oxide synthase 3, endothelial cell Mus musculus