PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22291917-0	2012	eNOS protects from atherosclerosis despite relevant superoxide production by the enzyme in apoE mice.	Superoxides	52-62	nitric oxide synthase 3, endothelial cell	Mus musculus	0-4	
22291917-5	2012	Here, we evaluate how endothelial nitric oxide synthase (eNOS) modulates leukocyte/endothelial- (L/E) and platelet/endothelial- (P/E) interactions in atherosclerosis and the production of nitric oxide (NO) and superoxide by the enzyme.	Superoxides	210-220	nitric oxide synthase 3, endothelial cell	Mus musculus	22-55	
22291917-5	2012	Here, we evaluate how endothelial nitric oxide synthase (eNOS) modulates leukocyte/endothelial- (L/E) and platelet/endothelial- (P/E) interactions in atherosclerosis and the production of nitric oxide (NO) and superoxide by the enzyme.	Superoxides	210-220	nitric oxide synthase 3, endothelial cell	Mus musculus	57-61	
22291917-9	2012	Pharmacological inhibition and genetic deletion of eNOS reduced vascular superoxide production, indicating uncoupling of the enzyme in apoE(-/-) vessels.	Superoxides	73-83	nitric oxide synthase 3, endothelial cell	Mus musculus	51-55	
22291917-10	2012	CONCLUSION: Overt plaque formation, increased vascular inflammation and L/E- interactions are associated with significant reduction of superoxide production in apoE(-/-)/eNOS(-/-) vessels.	Superoxides	135-145	nitric oxide synthase 3, endothelial cell	Mus musculus	170-174