Title : Hindbrain neuroglucopenia elicits site-specific transcriptional activation of glutamate decarboxylase-immunopositive neurons in the septopreoptic area of female rat brain.

Pub. Date : 2008

PMID : 17934249






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1 Since Fos immunolabeling of the SPO of rats treated by caudal fourth ventricular (CV4) administration of the glucose antimetabolite, 5-thioglucose (5TG), parallels the distribution of GABA neuronal perikarya, the current studies investigated the genomic responsiveness of neuroanatomically-defined populations of glutamate decarboxylase (GAD)-immunoreactive (-ir) neurons in this region of the brain to hindbrain glucoprivation. Glucose Fos proto-oncogene, AP-1 transcription factor subunit Rattus norvegicus