Title : Ubiquitin C-terminal hydrolase L1 (UCHL1) regulates post-myocardial infarction cardiac fibrosis through glucose-regulated protein of 78 kDa (GRP78).

Pub. Date : 2020 Jun 30

PMID : 32606430






3 Functional Relationships(s)
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1 Furthermore, we found that GRP78 was upregulated after UCHL1 knockdown and that the GRP78 inhibitor HA15 diminished the antifibrotic function exerted by UCHL1 knockdown in CFs stimulated with TGF-beta1. HA15 heat shock protein family A (Hsp70) member 5 Homo sapiens
2 Furthermore, we found that GRP78 was upregulated after UCHL1 knockdown and that the GRP78 inhibitor HA15 diminished the antifibrotic function exerted by UCHL1 knockdown in CFs stimulated with TGF-beta1. HA15 ubiquitin C-terminal hydrolase L1 Homo sapiens
3 Furthermore, we found that GRP78 was upregulated after UCHL1 knockdown and that the GRP78 inhibitor HA15 diminished the antifibrotic function exerted by UCHL1 knockdown in CFs stimulated with TGF-beta1. HA15 transforming growth factor beta 1 Homo sapiens