Title : HSV-1/TLR9-Mediated IFNβ and TNFα Induction Is Mal-Dependent in Macrophages.

Pub. Date : 2020

PMID : 31851971






3 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 We show for the first time that Mal is engaged in TLR9-de-pendent expression of genes encoding IFNbeta and TNFalpha in HSV-1-infected or CpG-C-treated macrophages and requires a noncanonical NF-kappaB pathway. cytidylyl-(3'-5')-cytidine mal, T cell differentiation protein Homo sapiens
2 We show for the first time that Mal is engaged in TLR9-de-pendent expression of genes encoding IFNbeta and TNFalpha in HSV-1-infected or CpG-C-treated macrophages and requires a noncanonical NF-kappaB pathway. cytidylyl-(3'-5')-cytidine toll like receptor 9 Homo sapiens
3 We show for the first time that Mal is engaged in TLR9-de-pendent expression of genes encoding IFNbeta and TNFalpha in HSV-1-infected or CpG-C-treated macrophages and requires a noncanonical NF-kappaB pathway. cytidylyl-(3'-5')-cytidine interferon beta 1 Homo sapiens