Title : Proviral insertion in murine lymphomas 2 promotes stomach cancer progression by regulating apoptosis via reactive oxygen species-triggered endoplasmic reticulum stress.

Pub. Date : 2018 Nov 17

PMID : 30340823






4 Functional Relationships(s)
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1 In addition, our data showed that PIM2 silence induced reactive oxygen species (ROS) production, leading to the activation of c-Jun N-terminal kinase (JNK). Reactive Oxygen Species proviral integration site 2 Mus musculus
2 In addition, our data showed that PIM2 silence induced reactive oxygen species (ROS) production, leading to the activation of c-Jun N-terminal kinase (JNK). Reactive Oxygen Species proviral integration site 2 Mus musculus
3 Importantly, we found that PIM2 knockdown-induced apoptosis and ER stress could be abolished by reducing reactive oxygen species (ROS) generation. Reactive Oxygen Species proviral integration site 2 Mus musculus
4 Importantly, we found that PIM2 knockdown-induced apoptosis and ER stress could be abolished by reducing reactive oxygen species (ROS) generation. Reactive Oxygen Species proviral integration site 2 Mus musculus