Title : Essential roles of Akt/Snail pathway in microcystin-LR-induced tight junction toxicity in Sertoli cell.

Pub. Date : 2018 Feb

PMID : 29307602






5 Functional Relationships(s)
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1 The activated Akt/GSK-3beta and snail signaling pathway largely accounted for MC-LRinduced TJs toxicity, which could be partially reversed by snail siRNA interference or AKT chemical inhibitor in TM4 cells. microcystin thymoma viral proto-oncogene 1 Mus musculus
2 The activated Akt/GSK-3beta and snail signaling pathway largely accounted for MC-LRinduced TJs toxicity, which could be partially reversed by snail siRNA interference or AKT chemical inhibitor in TM4 cells. microcystin glycogen synthase kinase 3 beta Mus musculus
3 The activated Akt/GSK-3beta and snail signaling pathway largely accounted for MC-LRinduced TJs toxicity, which could be partially reversed by snail siRNA interference or AKT chemical inhibitor in TM4 cells. microcystin snail family zinc finger 1 Mus musculus
4 The activated Akt/GSK-3beta and snail signaling pathway largely accounted for MC-LRinduced TJs toxicity, which could be partially reversed by snail siRNA interference or AKT chemical inhibitor in TM4 cells. microcystin snail family zinc finger 1 Mus musculus
5 The activated Akt/GSK-3beta and snail signaling pathway largely accounted for MC-LRinduced TJs toxicity, which could be partially reversed by snail siRNA interference or AKT chemical inhibitor in TM4 cells. microcystin thymoma viral proto-oncogene 1 Mus musculus