Title : An ALPHA7 Nicotinic Acetylcholine Receptor Agonist (GTS-21) Promotes C2C12 Myonuclear Accretion in Association with Release of Interleukin-6 (IL-6) and Improves Survival in Burned Mice.

Pub. Date : 2017 Aug

PMID : 28282360






4 Functional Relationships(s)
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1 The in vitro findings suggest that GTS-21-induced IL-6 release from muscle is mediated via alpha7AChRs upstream of Stat-3 and -5 pathways and is associated with myonuclear accretion, possibly via MyoD and Pax7 expression. 3-(2,4-dimethoxybenzylidene)anabaseine interleukin 6 Mus musculus
2 The in vitro findings suggest that GTS-21-induced IL-6 release from muscle is mediated via alpha7AChRs upstream of Stat-3 and -5 pathways and is associated with myonuclear accretion, possibly via MyoD and Pax7 expression. 3-(2,4-dimethoxybenzylidene)anabaseine signal transducer and activator of transcription 3 Mus musculus
3 The in vitro findings suggest that GTS-21-induced IL-6 release from muscle is mediated via alpha7AChRs upstream of Stat-3 and -5 pathways and is associated with myonuclear accretion, possibly via MyoD and Pax7 expression. 3-(2,4-dimethoxybenzylidene)anabaseine myogenic differentiation 1 Mus musculus
4 The in vitro findings suggest that GTS-21-induced IL-6 release from muscle is mediated via alpha7AChRs upstream of Stat-3 and -5 pathways and is associated with myonuclear accretion, possibly via MyoD and Pax7 expression. 3-(2,4-dimethoxybenzylidene)anabaseine paired box 7 Mus musculus