Title : Differential Cav2.1 and Cav2.3 channel inhibition by baclofen and α-conotoxin Vc1.1 via GABAB receptor activation.

Pub. Date : 2014 Apr

PMID : 24688019






1 Functional Relationships(s)
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1 In contrast, baclofen or Vc1.1 inhibition of Cav2.3 channels was solely mediated through voltage-independent pathways that could be disrupted by pertussis toxin, guanosine 5"-[beta-thio]diphosphate trilithium salt, or the GABABR antagonist CGP55845. CGP 55845A calcium voltage-gated channel subunit alpha1 E Homo sapiens