Title : Receptor for advanced glycation end-products promotes premature senescence of proximal tubular epithelial cells via activation of endoplasmic reticulum stress-dependent p21 signaling.

Pub. Date : 2014 Jan

PMID : 24113348






3 Functional Relationships(s)
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1 In addition, the fraction of SA-beta-gal or cells in the G0G1 phase were enhanced in cultured mouse proximal tubular epithelial cells (PTECs) and the expression of RAGE, GRP78 and p21 was up-regulated by advanced glycation end-products (AGEs) in a dose- and time-dependent manner. 2-(2-quinolinyl)-1H-indene--1,3(2H)-dione-6'-sulfonic acid advanced glycosylation end product-specific receptor Mus musculus
2 In addition, the fraction of SA-beta-gal or cells in the G0G1 phase were enhanced in cultured mouse proximal tubular epithelial cells (PTECs) and the expression of RAGE, GRP78 and p21 was up-regulated by advanced glycation end-products (AGEs) in a dose- and time-dependent manner. 2-(2-quinolinyl)-1H-indene--1,3(2H)-dione-6'-sulfonic acid heat shock protein 5 Mus musculus
3 In addition, the fraction of SA-beta-gal or cells in the G0G1 phase were enhanced in cultured mouse proximal tubular epithelial cells (PTECs) and the expression of RAGE, GRP78 and p21 was up-regulated by advanced glycation end-products (AGEs) in a dose- and time-dependent manner. 2-(2-quinolinyl)-1H-indene--1,3(2H)-dione-6'-sulfonic acid cyclin-dependent kinase inhibitor 1A (P21) Mus musculus