Pub. Date : 2011 May 20
PMID : 21502544
4 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | PURPOSE: The addition of cetuximab to irinotecan, fluorouracil, and leucovorin (FOLFIRI) as first-line treatment for metastatic colorectal cancer (mCRC) was shown to reduce the risk of disease progression and increase the chance of response in patients with KRAS wild-type disease. | FOLFIRI regimen | KRAS proto-oncogene, GTPase | Homo sapiens |
2 | The addition of cetuximab to FOLFIRI in patients with KRAS wild-type disease resulted in significant improvements in overall survival (median, 23.5 v 20.0 months; hazard ratio [HR], 0.796; P = .0093), progression-free survival (median, 9.9 v 8.4 months; HR, 0.696; P = .0012), and response (rate 57.3% v 39.7%; odds ratio, 2.069; P < .001) compared with FOLFIRI alone. | FOLFIRI regimen | KRAS proto-oncogene, GTPase | Homo sapiens |
3 | KRAS mutation status was confirmed as a powerful predictive biomarker for the efficacy of cetuximab plus FOLFIRI. | FOLFIRI regimen | KRAS proto-oncogene, GTPase | Homo sapiens |
4 | CONCLUSION: The addition of cetuximab to FOLFIRI as first-line therapy improves survival in patients with KRAS wild-type mCRC. | FOLFIRI regimen | KRAS proto-oncogene, GTPase | Homo sapiens |