Title : Phorbol ester stimulation of RasGRP1 regulates the sodium-chloride cotransporter by a PKC-independent pathway.

Pub. Date : 2007 Dec 11

PMID : 18077438






7 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Phorbol ester stimulation of RasGRP1 regulates the sodium-chloride cotransporter by a PKC-independent pathway. Phorbol Esters RAS guanyl releasing protein 1 Homo sapiens
2 Here we describe how one of those alternate targets of DAG/PE effects, Ras guanyl-releasing protein 1 (RasGRP1), mediates the PE-induced suppression of function and the surface expression of NCC. Diglycerides RAS guanyl releasing protein 1 Homo sapiens
3 Here we describe how one of those alternate targets of DAG/PE effects, Ras guanyl-releasing protein 1 (RasGRP1), mediates the PE-induced suppression of function and the surface expression of NCC. Diglycerides RAS guanyl releasing protein 1 Homo sapiens
4 Here we describe how one of those alternate targets of DAG/PE effects, Ras guanyl-releasing protein 1 (RasGRP1), mediates the PE-induced suppression of function and the surface expression of NCC. Phorbol Esters RAS guanyl releasing protein 1 Homo sapiens
5 Here we describe how one of those alternate targets of DAG/PE effects, Ras guanyl-releasing protein 1 (RasGRP1), mediates the PE-induced suppression of function and the surface expression of NCC. Phorbol Esters RAS guanyl releasing protein 1 Homo sapiens
6 Here we describe how one of those alternate targets of DAG/PE effects, Ras guanyl-releasing protein 1 (RasGRP1), mediates the PE-induced suppression of function and the surface expression of NCC. Phorbol Esters RAS guanyl releasing protein 1 Homo sapiens
7 Here we describe how one of those alternate targets of DAG/PE effects, Ras guanyl-releasing protein 1 (RasGRP1), mediates the PE-induced suppression of function and the surface expression of NCC. Phorbol Esters RAS guanyl releasing protein 1 Homo sapiens