Pub. Date : 1992 May
PMID : 1348524
17 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | N-methyl-D-aspartate antagonists block fos-like protein expression induced via multiple signaling pathways in cultured cortical neurons. | N-Methylaspartate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 2 | c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide. | Glutamic Acid | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 3 | c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide. | Glutamic Acid | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 4 | c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide. | Phorbol Esters | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 5 | c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide. | Phorbol Esters | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 6 | c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide. | Zinc | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 7 | c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide. | Zinc | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 8 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | Glutamic Acid | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 9 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | N-Methylaspartate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 10 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | N-Methylaspartate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 11 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | Dizocilpine Maleate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 12 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | N-Methylaspartate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 13 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | 4-(3-phosphonopropyl)piperazin-2-carboxylic acid | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 14 | Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate. | 2-amino-7-phosphonoheptanoate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 15 | These antagonists partially block high K(+)-, phorbol ester-, Zn(2+)-, and VIP-induced c-fos mRNA expression, but have no effect on bFGF-induced c-fos mRNA expression. | Phorbol Esters | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 16 | These antagonists partially block high K(+)-, phorbol ester-, Zn(2+)-, and VIP-induced c-fos mRNA expression, but have no effect on bFGF-induced c-fos mRNA expression. | Zinc | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |
| 17 | It is possible that NMDA antagonists prevent cortical plasticity by blocking induction of the Fos protein that would normally be induced by neurotrophic factors, neurotransmitters, and neuromodulators. | N-Methylaspartate | Fos proto-oncogene, AP-1 transcription factor subunit | Homo sapiens |