PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1348524-0	1992	N-methyl-D-aspartate antagonists block fos-like protein expression induced via multiple signaling pathways in cultured cortical neurons.	N-Methylaspartate	0-20	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	39-42	
1348524-1	1992	c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide.	Glutamic Acid	85-94	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	0-5	
1348524-1	1992	c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide.	Glutamic Acid	85-94	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	15-18	
1348524-1	1992	c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide.	Phorbol Esters	105-118	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	0-5	
1348524-1	1992	c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide.	Phorbol Esters	105-118	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	15-18	
1348524-1	1992	c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide.	Zinc	152-156	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	0-5	
1348524-1	1992	c-fos mRNA and Fos-like protein(s) (FLP) are induced in cultured cortical neurons by glutamate, high K+, phorbol ester, basic fibroblast growth factor, Zn2+, and vasoactive intestinal peptide.	Zinc	152-156	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	15-18	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	Glutamic Acid	0-9	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	N-Methylaspartate	71-91	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	N-Methylaspartate	93-97	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	Dizocilpine Maleate	111-117	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	N-Methylaspartate	135-139	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	4-(3-phosphonopropyl)piperazin-2-carboxylic acid	153-201	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-2	1992	Glutamate induction of c-fos mRNA and FLP is blocked by noncompetitive N-methyl-D-aspartate (NMDA) antagonist, MK-801, and competitive NMDA antagonists, 4-(3-phosphonopropyl)piperazin-2-carboxylic acid and 2-amino-7-phosphonoheptanoate.	2-amino-7-phosphonoheptanoate	206-235	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	23-28	
1348524-3	1992	These antagonists partially block high K(+)-, phorbol ester-, Zn(2+)-, and VIP-induced c-fos mRNA expression, but have no effect on bFGF-induced c-fos mRNA expression.	Phorbol Esters	46-59	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	87-92	
1348524-3	1992	These antagonists partially block high K(+)-, phorbol ester-, Zn(2+)-, and VIP-induced c-fos mRNA expression, but have no effect on bFGF-induced c-fos mRNA expression.	Zinc	62-68	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	87-92	
1348524-7	1992	It is possible that NMDA antagonists prevent cortical plasticity by blocking induction of the Fos protein that would normally be induced by neurotrophic factors, neurotransmitters, and neuromodulators.	N-Methylaspartate	20-24	Fos proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	94-97