Title : Antioxidants attenuate myocyte apoptosis in the remote non-infarcted myocardium following large myocardial infarction.

Pub. Date : 2000 Feb

PMID : 10728389






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1 CONCLUSIONS: Long-term treatment with the antioxidants probucol and pyrrolidine dithiocarbamate attenuates oxidative stress, myocyte apoptosis, caspase-3 like activity and the expression of p53, bax and caspase-3 within RM in rats after a large myocardial infarction. Probucol caspase 3 Rattus norvegicus
2 CONCLUSIONS: Long-term treatment with the antioxidants probucol and pyrrolidine dithiocarbamate attenuates oxidative stress, myocyte apoptosis, caspase-3 like activity and the expression of p53, bax and caspase-3 within RM in rats after a large myocardial infarction. Probucol caspase 3 Rattus norvegicus
3 CONCLUSIONS: Long-term treatment with the antioxidants probucol and pyrrolidine dithiocarbamate attenuates oxidative stress, myocyte apoptosis, caspase-3 like activity and the expression of p53, bax and caspase-3 within RM in rats after a large myocardial infarction. pyrrolidine dithiocarbamic acid caspase 3 Rattus norvegicus
4 CONCLUSIONS: Long-term treatment with the antioxidants probucol and pyrrolidine dithiocarbamate attenuates oxidative stress, myocyte apoptosis, caspase-3 like activity and the expression of p53, bax and caspase-3 within RM in rats after a large myocardial infarction. pyrrolidine dithiocarbamic acid caspase 3 Rattus norvegicus