Title : Involvement of caspase-3-like protease in the mechanism of cell death following focally evoked limbic seizures.

Pub. Date : 2000 Mar

PMID : 10693954






3 Functional Relationships(s)
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1 The putatively selective caspase-3 inhibitor N-benzyloxycarbonyl-Asp(OMe)-Glu(OMe)-Val-Asp(OMe)-fluoromethyl ketone significantly improved neuronal survival bilaterally within the hippocampal CA3/CA4 subfields following seizures. n-benzyloxycarbonyl-asp(ome)-glu(ome) caspase 3 Rattus norvegicus
2 The putatively selective caspase-3 inhibitor N-benzyloxycarbonyl-Asp(OMe)-Glu(OMe)-Val-Asp(OMe)-fluoromethyl ketone significantly improved neuronal survival bilaterally within the hippocampal CA3/CA4 subfields following seizures. val-asp(ome) caspase 3 Rattus norvegicus
3 The putatively selective caspase-3 inhibitor N-benzyloxycarbonyl-Asp(OMe)-Glu(OMe)-Val-Asp(OMe)-fluoromethyl ketone significantly improved neuronal survival bilaterally within the hippocampal CA3/CA4 subfields following seizures. 1,3-Difluoroacetone caspase 3 Rattus norvegicus