Title : Inhibition of inositol(1,4,5)Trisphosphate generation by endothelin-1 during postischemic reperfusion: A novel antiarrhythmic mechanism.

Pub. Date : 1999 Feb 16

PMID : 9989970






1 Functional Relationships(s)
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1 In parallel studies, endothelin-1 suppressed the development of reperfusion arrhythmias initiated by either thrombin (ventricular fibrillation, 75% to 39%, n=16 to 18) or norepinephrine (83% to 8%, n=12 to 22) (P<0.01 in both cases). Norepinephrine endothelin 1 Rattus norvegicus