Title : Role of phospholipase C, protein kinase C, and calcium in VEGF-induced venular hyperpermeability.

Pub. Date : 1999 Feb

PMID : 9950855






4 Functional Relationships(s)
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1 We previously demonstrated that vascular endothelial growth factor (VEGF)-elicited increase in the permeability of coronary venules was blocked by the nitric oxide (NO) synthase inhibitor NG-monomethyl-L-arginine (L-NMMA). omega-N-Methylarginine vascular endothelial growth factor A Homo sapiens
2 We previously demonstrated that vascular endothelial growth factor (VEGF)-elicited increase in the permeability of coronary venules was blocked by the nitric oxide (NO) synthase inhibitor NG-monomethyl-L-arginine (L-NMMA). omega-N-Methylarginine vascular endothelial growth factor A Homo sapiens
3 We previously demonstrated that vascular endothelial growth factor (VEGF)-elicited increase in the permeability of coronary venules was blocked by the nitric oxide (NO) synthase inhibitor NG-monomethyl-L-arginine (L-NMMA). omega-N-Methylarginine vascular endothelial growth factor A Homo sapiens
4 We previously demonstrated that vascular endothelial growth factor (VEGF)-elicited increase in the permeability of coronary venules was blocked by the nitric oxide (NO) synthase inhibitor NG-monomethyl-L-arginine (L-NMMA). omega-N-Methylarginine vascular endothelial growth factor A Homo sapiens