Title : Excretion of GSSG and glutathione conjugates mediated by MRP1 and cMOAT/MRP2.

Pub. Date : 1998

PMID : 9875554






1 Functional Relationships(s)
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1 A comparison of the transport properties across the bile canalicular membrane in normal and mutant rats, whose cMOAT function is hereditarily defective, has shown that the physiologic role of cMOAT is to excrete LTC4, bilirubin glucuronides, 171-estradiol-170-D-glucuronide, and reduced folates. Bilirubin ATP binding cassette subfamily C member 2 Rattus norvegicus