Title : K-ras point mutations in cancerous and noncancerous biliary epithelium in patients with pancreaticobiliary maljunction.

Pub. Date : 1996 Apr 15

PMID : 8608574






1 Functional Relationships(s)
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1 Direct sequence analysis of the K-ras oncogene revealed the mutation at codon 12 substituting the wild-type glycine (GGT) for aspartic acid (GAT) in all cancerous lesions of patients with PBM. Aspartic Acid KRAS proto-oncogene, GTPase Homo sapiens