Title : Angiotensin-mediated phosphatidylcholine hydrolysis and protein kinase C activation in mesangial cells.

Pub. Date : 1993 Oct

PMID : 8238300






7 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 Angiotensin II (ANG II) in mesangial cells (MC) promotes phosphatidylinositol (PI) hydrolysis resulting in diacylglycerol (DAG)-mediated increases in protein kinase C (PKC) activity. Diglycerides angiotensinogen Homo sapiens
2 Angiotensin II (ANG II) in mesangial cells (MC) promotes phosphatidylinositol (PI) hydrolysis resulting in diacylglycerol (DAG)-mediated increases in protein kinase C (PKC) activity. Diglycerides angiotensinogen Homo sapiens
3 Angiotensin II (ANG II) in mesangial cells (MC) promotes phosphatidylinositol (PI) hydrolysis resulting in diacylglycerol (DAG)-mediated increases in protein kinase C (PKC) activity. Diglycerides angiotensinogen Homo sapiens
4 Angiotensin II (ANG II) in mesangial cells (MC) promotes phosphatidylinositol (PI) hydrolysis resulting in diacylglycerol (DAG)-mediated increases in protein kinase C (PKC) activity. Diglycerides angiotensinogen Homo sapiens
5 ANG II-mediated PC hydrolysis augmented DAG for 30 min. Diglycerides angiotensinogen Homo sapiens
6 Whereas downregulation prevented PLD-mediated PEt elevation, ANG II-stimulated DAG formation in myristate-labeled cells was unaltered, indicating PC-PLC activation. Diglycerides angiotensinogen Homo sapiens
7 ANG II mediated PC breakdown principally via PC-PLC provides a mechanism for maintaining elevated DAG levels and PKC activation. Diglycerides angiotensinogen Homo sapiens