Pub. Date : 1994 Apr 15
PMID : 8161775
1 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | We found that BCR-ABL expression inappropriately prolongs the growth factor-independent survival of CML myeloid progenitors and granulocytes by inhibiting apoptosis, a genetically programmed process of active cell death; inhibition of BCR-ABL expression by antisense oligonucleotides reversed the suppression of apoptosis as well as the enhancement of survival. | Oligonucleotides | ABL proto-oncogene 1, non-receptor tyrosine kinase | Homo sapiens |