Pub. Date : 1994 Dec
PMID : 7962129
8 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
| 1 | Results of previous studies show that the expression of fibronectin and its cell-surface fibronectin binding receptor is coregulated in 3-methylchloranthrene transformation of normal AKR-2B cells to form AKR-MCA cells and in N,N,-dimethylformamide (DMF) induction of differentiation of transformed AKR-MCA cells (1990, J. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 2 | Results of previous studies show that the expression of fibronectin and its cell-surface fibronectin binding receptor is coregulated in 3-methylchloranthrene transformation of normal AKR-2B cells to form AKR-MCA cells and in N,N,-dimethylformamide (DMF) induction of differentiation of transformed AKR-MCA cells (1990, J. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 3 | Results of previous studies show that the expression of fibronectin and its cell-surface fibronectin binding receptor is coregulated in 3-methylchloranthrene transformation of normal AKR-2B cells to form AKR-MCA cells and in N,N,-dimethylformamide (DMF) induction of differentiation of transformed AKR-MCA cells (1990, J. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 4 | Results of previous studies show that the expression of fibronectin and its cell-surface fibronectin binding receptor is coregulated in 3-methylchloranthrene transformation of normal AKR-2B cells to form AKR-MCA cells and in N,N,-dimethylformamide (DMF) induction of differentiation of transformed AKR-MCA cells (1990, J. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 5 | Expression of antisense fibronectin RNA in AKR-MCA cells down-modulated the ability of DMF to restore normal fibronectin production, cell-surface fibronectin binding receptor, adhesion to extracellular fibronectin, and cellular morphology. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 6 | Expression of antisense fibronectin RNA in AKR-MCA cells down-modulated the ability of DMF to restore normal fibronectin production, cell-surface fibronectin binding receptor, adhesion to extracellular fibronectin, and cellular morphology. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 7 | Expression of antisense fibronectin RNA in AKR-MCA cells down-modulated the ability of DMF to restore normal fibronectin production, cell-surface fibronectin binding receptor, adhesion to extracellular fibronectin, and cellular morphology. | Dimethylformamide | fibronectin 1 | Mus musculus |
| 8 | Expression of antisense fibronectin RNA in AKR-MCA cells down-modulated the ability of DMF to restore normal fibronectin production, cell-surface fibronectin binding receptor, adhesion to extracellular fibronectin, and cellular morphology. | Dimethylformamide | fibronectin 1 | Mus musculus |