Title : Attenuation of the induction of nitric oxide synthase by endogenous glucocorticoids accounts for endotoxin tolerance in vivo.

Pub. Date : 1994 Jan 4

PMID : 7506416






2 Functional Relationships(s)
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Compound Name
Protein Name
Organism
1 An enhanced formation of nitric oxide (NO) due to induction of a calcium-independent (inducible) NO synthase (iNOS) contributes importantly to the cardiovascular failure caused by bacterial endotoxin. Calcium nitric oxide synthase 2 Rattus norvegicus
2 An enhanced formation of nitric oxide (NO) due to induction of a calcium-independent (inducible) NO synthase (iNOS) contributes importantly to the cardiovascular failure caused by bacterial endotoxin. Calcium nitric oxide synthase 2 Rattus norvegicus