Title : Increased apoptotic sensitivity of glioblastoma enables therapeutic targeting by BH3-mimetics.

Pub. Date : 2022 Apr 26

PMID : 35473984






1 Functional Relationships(s)
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1 These data demonstrate that BCL-xL and MCL-1 pro-survival function is a fundamental prerequisite for GBM survival that can be therapeutically exploited by BH3-mimetics. BH 3 MCL1 apoptosis regulator, BCL2 family member Homo sapiens