Title : Vitamin D3 Protects Mice from Diquat-Induced Oxidative Stress through the NF-κB/Nrf2/HO-1 Signaling Pathway.

Pub. Date : 2021

PMID : 34824670






3 Functional Relationships(s)
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1 Vitamin D3 Protects Mice from Diquat-Induced Oxidative Stress through the NF-kappaB/Nrf2/HO-1 Signaling Pathway. Cholecalciferol nuclear factor, erythroid derived 2, like 2 Mus musculus
2 In addition, the results indicated that vitamin D3 could significantly reduce the phosphorylation level of NF-kappaB (p65) and enhance the expression of Nrf2 and HO-1 in the jejunum compared with the diquat-induced group. Cholecalciferol nuclear factor, erythroid derived 2, like 2 Mus musculus
3 This study suggested that oral administration of vitamin D3 can protect mice against oxidative damage by inhibiting the phosphorylation level of NF-kappaB (p65) and activating Nrf2-related signaling pathways. Cholecalciferol nuclear factor, erythroid derived 2, like 2 Mus musculus