PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34824670-0	2021	Vitamin D3 Protects Mice from Diquat-Induced Oxidative Stress through the NF-kappaB/Nrf2/HO-1 Signaling Pathway.	Cholecalciferol	0-10	nuclear factor, erythroid derived 2, like 2	Mus musculus	84-88	
34824670-7	2021	In addition, the results indicated that vitamin D3 could significantly reduce the phosphorylation level of NF-kappaB (p65) and enhance the expression of Nrf2 and HO-1 in the jejunum compared with the diquat-induced group.	Cholecalciferol	40-50	nuclear factor, erythroid derived 2, like 2	Mus musculus	153-157	
34824670-8	2021	This study suggested that oral administration of vitamin D3 can protect mice against oxidative damage by inhibiting the phosphorylation level of NF-kappaB (p65) and activating Nrf2-related signaling pathways.	Cholecalciferol	49-59	nuclear factor, erythroid derived 2, like 2	Mus musculus	176-180