Title : Autophagy inhibition facilitates wound closure partially dependent on the YAP/IL-33 signaling in a mouse model of skin wound healing.

Pub. Date : 2021 Oct

PMID : 34547141






1 Functional Relationships(s)
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1 Herein, treating with 3-MA markedly suppressed the expression of tumor necrosis factor-alpha (TNF-alpha), IL-1beta, and IL-6, promoted that of IL-10, IL-33, and ST2, while RAP administration reverted SWH-induced the up-regulation of these inflammatory cytokines mentioned above. 3-methyladenine interleukin 6 Mus musculus