Title : Integration of data from the cell-based ERK1/2 ELISA and the Comparative Toxicogenomics Database deciphers the potential mode of action of bisphenol A and benzo[a]pyrene in lung neoplasm.

Pub. Date : 2021 Dec

PMID : 34329126






5 Functional Relationships(s)
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1 Integration of data from the cell-based ERK1/2 ELISA and the Comparative Toxicogenomics Database deciphers the potential mode of action of bisphenol A and benzo(a)pyrene in lung neoplasm. Benzo(a)pyrene mitogen-activated protein kinase 3 Homo sapiens
2 A549 cells were exposed to bisphenol A (BPA), benzo(a)pyrene (BaP), tributyltin (TBT), and ibuprofen from 10-12 M to 10-5 M. Results show that BPA, BaP, and TBT can activate ERK1/2 in A549 cells. Benzo(a)pyrene mitogen-activated protein kinase 3 Homo sapiens
3 A549 cells were exposed to bisphenol A (BPA), benzo(a)pyrene (BaP), tributyltin (TBT), and ibuprofen from 10-12 M to 10-5 M. Results show that BPA, BaP, and TBT can activate ERK1/2 in A549 cells. Benzo(a)pyrene mitogen-activated protein kinase 3 Homo sapiens
4 A549 cells were exposed to bisphenol A (BPA), benzo(a)pyrene (BaP), tributyltin (TBT), and ibuprofen from 10-12 M to 10-5 M. Results show that BPA, BaP, and TBT can activate ERK1/2 in A549 cells. Benzo(a)pyrene mitogen-activated protein kinase 3 Homo sapiens
5 We selected BPA and BaP to elucidate the molecular events connecting chemical exposure, ERK1/2 signaling, phenotypes, and lung neoplasm (LN) using CTD. Benzo(a)pyrene mitogen-activated protein kinase 3 Homo sapiens