Title : NOX4-Derived ROS Mediates TGF-β1-Induced Metabolic Reprogramming during Epithelial-Mesenchymal Transition through the PI3K/AKT/HIF-1α Pathway in Glioblastoma.

Pub. Date : 2021

PMID : 34257808






2 Functional Relationships(s)
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1 NOX4-Derived ROS Mediates TGF-beta1-Induced Metabolic Reprogramming during Epithelial-Mesenchymal Transition through the PI3K/AKT/HIF-1alpha Pathway in Glioblastoma. Reactive Oxygen Species hypoxia inducible factor 1 subunit alpha Homo sapiens
2 Moreover, TGF-beta1-induced metabolic reprogramming and EMT which modulated by NOX4/ROS were blocked when the phosphoinositide3-kinase (PI3K)/AKT/HIF-1alpha signaling pathways were inhibited. Reactive Oxygen Species hypoxia inducible factor 1 subunit alpha Homo sapiens