Title : Organ-specific cholesterol metabolic aberration fuels liver metastasis of colorectal cancer.

Pub. Date : 2021

PMID : 33995676






5 Functional Relationships(s)
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1 The role of SREBP2-dependent cholesterol biosynthesis pathway in cell growth and CRC liver metastasis were determined by SREBP2 silencing in CRC cell lines and experimental metastasis models including, intra-splenic injection models and liver orthotropic injection model. Cholesterol sterol regulatory element binding transcription factor 2 Homo sapiens
2 The role of SREBP2-dependent cholesterol biosynthesis pathway in cell growth and CRC liver metastasis were determined by SREBP2 silencing in CRC cell lines and experimental metastasis models including, intra-splenic injection models and liver orthotropic injection model. Cholesterol sterol regulatory element binding transcription factor 2 Homo sapiens
3 Results: In the present study, we identify a colorectal cancer (CRC) liver metastasis-specific cholesterol metabolic pathway involving the activation of SREBP2-dependent cholesterol biosynthesis, which is required for the colonization and growth of metastatic CRC cells in the liver. Cholesterol sterol regulatory element binding transcription factor 2 Homo sapiens
4 Results: In the present study, we identify a colorectal cancer (CRC) liver metastasis-specific cholesterol metabolic pathway involving the activation of SREBP2-dependent cholesterol biosynthesis, which is required for the colonization and growth of metastatic CRC cells in the liver. Cholesterol sterol regulatory element binding transcription factor 2 Homo sapiens
5 Mechanically, hepatocyte growth factor (HGF) from liver environment activates SREBP2-dependent cholesterol biosynthesis pathway by activating c-Met/PI3K/AKT/mTOR axis in CRC cells. Cholesterol sterol regulatory element binding transcription factor 2 Homo sapiens