Title : Rotor Syndrome: Glucuronidated Bile Acidemia From Defective Reuptake by Hepatocytes.

Pub. Date : 2021 Apr

PMID : 33860121






1 Functional Relationships(s)
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1 In contrast but similarly to healthy controls, only trace amounts of BA-3G were detected in patients with constitutional indocyanine green excretory defect (OATP1B3 deficiency) or sodium-taurocholate cotransporting polypeptide (NTCP; gene, solute carrier family 10 member 1 [SLC10A1]) deficiency. Bile Acids and Salts solute carrier organic anion transporter family member 1B3 Homo sapiens