Title : Nicotine Synergizes with High-Fat Diet to Induce an Anti-Inflammatory Microenvironment to Promote Breast Tumor Growth.

Pub. Date : 2020

PMID : 33414685






4 Functional Relationships(s)
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1 Scratch-wound assay showed marked reduction in proliferation/migration of nicotine and palmitate-treated breast cancer cells with mecamylamine (MEC), a nicotine acetylcholine receptor (nAchR) antagonist. Mecamylamine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
2 Scratch-wound assay showed marked reduction in proliferation/migration of nicotine and palmitate-treated breast cancer cells with mecamylamine (MEC), a nicotine acetylcholine receptor (nAchR) antagonist. Mecamylamine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
3 Scratch-wound assay showed marked reduction in proliferation/migration of nicotine and palmitate-treated breast cancer cells with mecamylamine (MEC), a nicotine acetylcholine receptor (nAchR) antagonist. Mecamylamine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
4 Scratch-wound assay showed marked reduction in proliferation/migration of nicotine and palmitate-treated breast cancer cells with mecamylamine (MEC), a nicotine acetylcholine receptor (nAchR) antagonist. Mecamylamine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus