Title : CASK, APBA1, and STXBP1 collaborate during insulin secretion.

Pub. Date : 2021 Jan 15

PMID : 33159991






1 Functional Relationships(s)
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1 High fatty acid stimulation decreased insulin secretion along with CASK, APBA1, and STXBP1 expression; Cask overexpression enhanced CASK/APBA1/STXBP1 tripartite complex function, and may thereby rescue lipotoxicity-induced insulin-release defects. Fatty Acids insulin Homo sapiens