Title : PPARα Inhibition Overcomes Tumor-Derived Exosomal Lipid-Induced Dendritic Cell Dysfunction.

Pub. Date : 2020 Oct 20

PMID : 33086073






2 Functional Relationships(s)
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1 Mechanistically, peroxisome proliferator activated receptor (PPAR) alpha responds to the fatty acids delivered by TDEs, resulting in excess lipid droplet biogenesis and enhanced fatty acid oxidation (FAO), culminating in a metabolic shift toward mitochondrial oxidative phosphorylation, which drives DC immune dysfunction. Fatty Acids peroxisome proliferator activated receptor alpha Homo sapiens
2 Mechanistically, peroxisome proliferator activated receptor (PPAR) alpha responds to the fatty acids delivered by TDEs, resulting in excess lipid droplet biogenesis and enhanced fatty acid oxidation (FAO), culminating in a metabolic shift toward mitochondrial oxidative phosphorylation, which drives DC immune dysfunction. Fatty Acids peroxisome proliferator activated receptor alpha Homo sapiens