Pub. Date : 2020 Aug 27
PMID : 32736702
4 Functional Relationships(s)Download |
Sentence | Compound Name | Protein Name | Organism |
1 | TGF-beta1 treatment increased reactive oxygen species (ROS) via NOX4 upregulation, which acts downstream of ERK and mTORC1, as the ROS scavenger N-acetylcysteine and a pan-NADPH oxidase (NOX) inhibitor DPI dissipated excess ROS generation. | Reactive Oxygen Species | mitogen-activated protein kinase 1 | Homo sapiens |
2 | TGF-beta1 treatment increased reactive oxygen species (ROS) via NOX4 upregulation, which acts downstream of ERK and mTORC1, as the ROS scavenger N-acetylcysteine and a pan-NADPH oxidase (NOX) inhibitor DPI dissipated excess ROS generation. | Reactive Oxygen Species | mitogen-activated protein kinase 1 | Homo sapiens |
3 | TGF-beta1 treatment increased reactive oxygen species (ROS) via NOX4 upregulation, which acts downstream of ERK and mTORC1, as the ROS scavenger N-acetylcysteine and a pan-NADPH oxidase (NOX) inhibitor DPI dissipated excess ROS generation. | Reactive Oxygen Species | mitogen-activated protein kinase 1 | Homo sapiens |
4 | TGF-beta1 treatment increased reactive oxygen species (ROS) via NOX4 upregulation, which acts downstream of ERK and mTORC1, as the ROS scavenger N-acetylcysteine and a pan-NADPH oxidase (NOX) inhibitor DPI dissipated excess ROS generation. | Reactive Oxygen Species | mitogen-activated protein kinase 1 | Homo sapiens |