Title : Acute nicotine administration stimulates ciliary activity via α3β4 nAChR in the mouse trachea.

Pub. Date : 2020 Jul

PMID : 32304995






4 Functional Relationships(s)
Download
Sentence
Compound Name
Protein Name
Organism
1 Acute nicotine administration stimulates ciliary activity via alpha3beta4 nAChR in the mouse trachea. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
2 Nicotine-induced PTS acceleration was sensitive to the general nAChR inhibitors mecamylamine and d-tubocurarine as well as to the alpha3beta4-nAChR antagonist alpha-conotoxin AulB, but not to other antagonists primarily addressing alpha3beta2-nAChR or alpha4-, alpha7- and alpha9-containing nAChR. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
3 Nicotine-induced PTS acceleration was sensitive to the general nAChR inhibitors mecamylamine and d-tubocurarine as well as to the alpha3beta4-nAChR antagonist alpha-conotoxin AulB, but not to other antagonists primarily addressing alpha3beta2-nAChR or alpha4-, alpha7- and alpha9-containing nAChR. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus
4 Nicotine-induced PTS acceleration was sensitive to the general nAChR inhibitors mecamylamine and d-tubocurarine as well as to the alpha3beta4-nAChR antagonist alpha-conotoxin AulB, but not to other antagonists primarily addressing alpha3beta2-nAChR or alpha4-, alpha7- and alpha9-containing nAChR. Nicotine cholinergic receptor, nicotinic, alpha polypeptide 7 Mus musculus