Title : ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKα2.

Pub. Date : 2020 May

PMID : 32244181






6 Functional Relationships(s)
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1 ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKalpha2. Glucose apolipoprotein A-I Mus musculus
2 ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKalpha2. Glucose apolipoprotein A-I Mus musculus
3 OBJECTIVE: Acute administration of the main protein component of high-density lipoprotein, apolipoprotein A-I (ApoA-1), improves glucose uptake in skeletal muscle. Glucose apolipoprotein A-I Mus musculus
4 OBJECTIVE: Acute administration of the main protein component of high-density lipoprotein, apolipoprotein A-I (ApoA-1), improves glucose uptake in skeletal muscle. Glucose apolipoprotein A-I Mus musculus
5 We therefore investigated whether AMPK is necessary for ApoA-1-stimulated glucose uptake in intact heart and skeletal muscle in vivo. Glucose apolipoprotein A-I Mus musculus
6 CONCLUSIONS: In conclusion, ApoA-1 stimulates in vivo glucose disposal into skeletal and heart muscle independently of AMPKalpha2. Glucose apolipoprotein A-I Mus musculus