PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32244181-0	2020	ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKalpha2.	Glucose	16-23	apolipoprotein A-I	Mus musculus	0-6	
32244181-0	2020	ApoA-1 improves glucose tolerance by increasing glucose uptake into heart and skeletal muscle independently of AMPKalpha2.	Glucose	48-55	apolipoprotein A-I	Mus musculus	0-6	
32244181-1	2020	OBJECTIVE: Acute administration of the main protein component of high-density lipoprotein, apolipoprotein A-I (ApoA-1), improves glucose uptake in skeletal muscle.	Glucose	129-136	apolipoprotein A-I	Mus musculus	91-109	
32244181-1	2020	OBJECTIVE: Acute administration of the main protein component of high-density lipoprotein, apolipoprotein A-I (ApoA-1), improves glucose uptake in skeletal muscle.	Glucose	129-136	apolipoprotein A-I	Mus musculus	111-117	
32244181-3	2020	We therefore investigated whether AMPK is necessary for ApoA-1-stimulated glucose uptake in intact heart and skeletal muscle in vivo.	Glucose	74-81	apolipoprotein A-I	Mus musculus	56-62	
32244181-12	2020	CONCLUSIONS: In conclusion, ApoA-1 stimulates in vivo glucose disposal into skeletal and heart muscle independently of AMPKalpha2.	Glucose	54-61	apolipoprotein A-I	Mus musculus	28-34