Title : GluA1 in Central Amygdala Promotes Opioid Use and Reverses Inhibitory Effect of Pain.

Pub. Date : 2020 Feb 1

PMID : 31863796






3 Functional Relationships(s)
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Protein Name
Organism
1 Microinjection of NASPM, a selective inhibitor of homomeric GluA1-AMPARs, into CeA inhibited morphine intake. Morphine glutamate ionotropic receptor AMPA type subunit 1 Rattus norvegicus
2 Furthermore, viral overexpression of GluA1 protein in CeA maintained morphine intake at a higher level than controls and reversed the pain-induced reduction in morphine intake. Morphine glutamate ionotropic receptor AMPA type subunit 1 Rattus norvegicus
3 Furthermore, viral overexpression of GluA1 protein in CeA maintained morphine intake at a higher level than controls and reversed the pain-induced reduction in morphine intake. Morphine glutamate ionotropic receptor AMPA type subunit 1 Rattus norvegicus