Title : Celastrol, a plant-derived triterpene, induces cisplatin-resistance nasopharyngeal carcinoma cancer cell apoptosis though ERK1/2 and p38 MAPK signaling pathway.

Pub. Date : 2019 May

PMID : 31022663






4 Functional Relationships(s)
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1 Celastrol, a plant-derived triterpene, induces cisplatin-resistance nasopharyngeal carcinoma cancer cell apoptosis though ERK1/2 and p38 MAPK signaling pathway. celastrol mitogen-activated protein kinase 3 Homo sapiens
2 Celastrol, a plant-derived triterpene, induces cisplatin-resistance nasopharyngeal carcinoma cancer cell apoptosis though ERK1/2 and p38 MAPK signaling pathway. celastrol mitogen-activated protein kinase 3 Homo sapiens
3 With further analysis, we observed that celastrol-induced activation of caspases was accompanied by increased phosphorylation of MAPK pathway proteins, p38, ERK1/2. celastrol mitogen-activated protein kinase 3 Homo sapiens
4 With further analysis, we observed that celastrol-induced activation of caspases was accompanied by increased phosphorylation of MAPK pathway proteins, p38, ERK1/2. celastrol mitogen-activated protein kinase 3 Homo sapiens